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In a study rats the dose of Myo-inositol (50mg/kg oral dose) was sufficient to induce a dose-dependent increase in the levels of enzyme activity, and to suppress the expression of cytoprotective stress proteins.
This was followed by a decrease in mitochondrial DNA content and an increase in the expression of cytochrome c oxidase, a gene known to azithromycin 250 online be involved in the induction of apoptosis.
These effects were associated with a decrease in the expression of proteins involved in protein folding and synthesis, as well DNA damage.
Mitochondrial dysfunction in Myo-inositol-induced cytoprotection
These results suggest that mitochondrial dysfunction and oxidative stress in the presence of Myo-inositol may be a potent mechanism of resistance to oxidative stress, particularly given the effects of this compound on other stress-related proteins.
Moenin
An acetylated derivative of Myo-inositol and a naturally occurring substance present in certain foods, Moenin has also been found in human breast milk and cancer cells.
It has been seen to have a protective effect against breast cancer by inhibiting the proliferation of tumor cells.
This has been tested in vitro and vivo it acts via a cysteine-mediated mechanism, which inhibits the expression of breast cancer cell growth characteristics, and inactivates the of normal breast cancer cells.
As a result of this, Moenin inhibits cell proliferation azithromycin online bestellen and increases apoptosis in vitro vivo.
It's effects are less well studied, but Moenin appears to have neuroprotective effects in rodents and humans, both of which have been used to date as a screening a